Background Recent evidence has shown that several chemokines—including those
involved in angiogenesis—have been implicated in the pathogenesis of idiopathic
pulmonary fibrosis (IPF) and sarcoidosis. We speculated that these differences could be
attributed to distinct angiogenic and angiostatic profiles. This hypothesis was investigated by
estimating the levels of three angiogenic chemokines (growth-related gene [GRO]-α,
epithelial neutrophil-activating protein [ENA]-78, and interleukin [IL]-8), and three angiostatic …